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In some cases, alternative treatments with fewer metabolic effects might be appropriate, or the timing of therapies could be optimised to minimise conflicting effects whilst maintaining therapeutic benefit
International Journal of Neuropsychopharmacology , 962-977
El ALC se sintetiza en las mitocondrias y puede participar en el metabolismo energtico, particularmente en el cerebro
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ER signaling helps protect against hepatic steatosis and fibrosis by activating pathways that reduce oxidative stress and inhibit pro-inflammatory cytokines such as TNF- and IL-6[66,67]
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